Induction of diabetes in the RIP-B7. 1 mouse model is critically dependent on TLR3 and MyD88 pathways and is associated with alterations in the intestinal …
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - Diabetes, 2014 - Am Diabetes Assoc
RIP-B7. 1 transgenic mice express B7. 1 costimulatory molecules in pancreatic islets and
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
Induction of diabetes in the RIP-B7. 1 mouse model is critically dependent on TLR3 and MyD88 pathways and is associated with alterations in the intestinal …
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - …, 2014 - pubmed.ncbi.nlm.nih.gov
RIP-B7. 1 transgenic mice express B7. 1 costimulatory molecules in pancreatic islets and
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
Induction of diabetes in the RIP-B7. 1 mouse model is critically dependent on TLR3 and MyD88 pathways and is associated with alterations in the intestinal …
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - Diabetes, 2013 - europepmc.org
RIP-B7. 1 transgenic mice express B7. 1 costimulatory molecules in pancreatic islets and
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
[PDF][PDF] Diabetes Publish Ahead of Print, published online December 18, 2013
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - 2014 - researchgate.net
The innate immune system uses pattern-recognition receptors, such as Toll-like receptors
(TLRs), nucleotidebinding oligomerization domain (NOD)-like receptors, and retinoic acid …
(TLRs), nucleotidebinding oligomerization domain (NOD)-like receptors, and retinoic acid …
Induction of Diabetes in the RIP-B7. 1 Mouse Model Is Critically Dependent on TLR3 and MyD88 Pathways and Is Associated With Alterations in the Intestinal …
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - …, 2014 - search.ebscohost.com
Abstract RIP-B7. 1 transgenic mice express B7. 1 costimulatory molecules in pancreatic
islets and develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a …
islets and develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a …
Induction of diabetes in the RIP-B7. 1 mouse model is critically dependent on TLR3 and MyD88 pathways and is associated with alterations in the intestinal …
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - Diabetes, 2014 - go.gale.com
RIP-B7. 1 transgenic mice express B7. 1 costimulatory molecules in pancreatic islets and
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
develop diabetes after treatment with polyinosinic: polycytidylic acid (poly I: C), a synthetic …
Induction of Diabetes in the RIP-B7. 1 Mouse Model Is Critically Dependent on TLR3 and MyD88 Pathways and Is Associated With Alterations in the Intestinal …
AK Alkanani, N Hara, E Lien, D Ir, CV Kotter… - Diabetes, 2014 - cir.nii.ac.jp
抄録< jats: p> RIP-B7. 1 transgenic mice express B7. 1 costimulatory molecules in
pancreatic islets and develop diabetes after treatment with polyinosinic: polycytidylic acid …
pancreatic islets and develop diabetes after treatment with polyinosinic: polycytidylic acid …
[引用][C] Induction of Diabetes in the RIP-B7. 1 Mouse Model Is Critically Dependent on TLR3 and MyD88 Pathways and Is Associated With Alterations in the Intestinal …
AK ALKANANI, N HARA, E LIEN, D IR… - … (New York, NY), 2014 - pascal-francis.inist.fr
Induction of Diabetes in the RIP-B7.1 Mouse Model Is Critically Dependent on TLR3 and
MyD88 Pathways and Is Associated With Alterations in the Intestinal Microbiome CNRS Inist …
MyD88 Pathways and Is Associated With Alterations in the Intestinal Microbiome CNRS Inist …