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[HTML][HTML] Oxidative stress induces nuclear-to-cytosol shift of hMSH3, a potential mechanism for EMAST in colorectal cancer cells

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - PloS one, 2012 - journals.plos.org
Background Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …
被引用次数：62 相关文章

Oxidative stress induces nuclear-to-cytosol shift of hMSH3, a potential mechanism for EMAST in colorectal cancer cells

SS Tseng-Rogenski, H Chung, MB Wilk… - PloS …, 2012 - pubmed.ncbi.nlm.nih.gov
Background Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …

Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST in Colorectal Cancer Cells

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - PLoS ONE, 2012 - go.gale.com
Background Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …

[引用][C] Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST in Colorectal Cancer Cells

SS Tseng-Rogenski, H Chung, MB Wilk… - PLoS …, 2012 - ui.adsabs.harvard.edu
Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST
in Colorectal Cancer Cells - NASA/ADS Now on home page ads icon ads Enable full ADS view …

Oxidative stress induces nuclear-to-cytosol shift of hMSH3, a potential mechanism for EMAST in colorectal cancer cells.

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - Plos one, 2012 - europepmc.org
Background Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …

[引用][C] Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST in Colorectal Cancer Cells

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - PLoS ONE, 2012 - cir.nii.ac.jp
Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for
EMAST in Colorectal Cancer Cells | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ …
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[HTML][HTML] Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST in Colorectal Cancer Cells

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - PLoS ONE, 2012 - ncbi.nlm.nih.gov
Background Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …

Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential Mechanism for EMAST in Colorectal Cancer Cells.

SS Tseng-Rogenski, H Chung, MB Wilk… - PLoS …, 2012 - search.ebscohost.com
Background: Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …
[PDF] psu.edu

[PDF][PDF] Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - 2012 - Citeseer
Background: Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …
[PDF] scienceopen.com

[PDF][PDF] Oxidative Stress Induces Nuclear-to-Cytosol Shift of hMSH3, a Potential

SS Tseng-Rogenski, H Chung, MB Wilk, S Zhang… - 2012 - scienceopen.com
Background: Elevated microsatellite alterations at selected tetranucleotide repeats (EMAST)
is a genetic signature observed in 60% of sporadic colorectal cancers (CRCs). Unlike …