IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP signaling …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - Molecular …, 2013 - Elsevier
Background Complement component C5-derived C5a locally generated in the brain has
been shown to protect against glutamate-induced neuronal apoptosis and beta-amyloid (Aβ) …
被引用次数：51 相关文章

IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP signaling …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - Molecular …, 2013 - infona.pl
Complement component C5-derived C5a locally generated in the brain has been shown to
protect against glutamate-induced neuronal apoptosis and beta-amyloid (Aβ) toxicity, but the …

IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP signaling …

B Gong, Y Pan, W Zhao, L Knable… - Molecular …, 2013 - pubmed.ncbi.nlm.nih.gov
Background Complement component C5-derived C5a locally generated in the brain has
been shown to protect against glutamate-induced neuronal apoptosis and beta-amyloid (Aβ) …
[PDF] google.com

[PDF][PDF] IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - Molecular …, 2013 - drive.google.com
abstract Background: Complement component C5-derived C5a locally generated in the
brain has been shown to protect against glutamate-induced neuronal apoptosis and beta …
[PDF] academia.edu

[PDF][PDF] IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - Molecular …, 2013 - academia.edu
abstract Background: Complement component C5-derived C5a locally generated in the
brain has been shown to protect against glutamate-induced neuronal apoptosis and beta …

IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP signaling …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - Molecular …, 2013 - europepmc.org
Background Complement component C5-derived C5a locally generated in the brain has
been shown to protect against glutamate-induced neuronal apoptosis and beta-amyloid (Aβ) …
[PDF] academia.edu

[PDF][PDF] IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - Molecular …, 2013 - academia.edu
abstract Background: Complement component C5-derived C5a locally generated in the
brain has been shown to protect against glutamate-induced neuronal apoptosis and beta …

[引用][C] IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP …

B Gong, Y Pan, W Zhao, L Knable, P Vempati… - 2013