Recombinant Netrin-1 binding UNC5B receptor attenuates neuroinflammation and brain injury via PPARγ/NFκB signaling pathway after subarachnoid hemorrhage in …

Z Xie, L Huang, B Enkhjargal, C Reis, W Wan… - Brain, behavior, and …, 2018 - Elsevier
Neuroinflammation is an essential mechanism involved in the pathogenesis of
subarachnoid hemorrhage (SAH)-induced brain injury. Recently, Netrin-1 (NTN-1) is well …

Recombinant Netrin-1 binding UNC5B receptor attenuates neuroinflammation and brain injury via PPARγ/NFκB signaling pathway after subarachnoid hemorrhage in …

Z Xie, L Huang, B Enkhjargal, C Reis… - Brain, behavior …, 2018 - pubmed.ncbi.nlm.nih.gov
Neuroinflammation is an essential mechanism involved in the pathogenesis of
subarachnoid hemorrhage (SAH)-induced brain injury. Recently, Netrin-1 (NTN-1) is well …

[HTML][HTML] Recombinant Netrin-1 Binding UNC5B Receptor Attenuates Neuroinflammation and Brain Injury via PPARγ/NFκB Signaling Pathway after Subarachnoid …

Z Xie, L Huang, B Enkhjargal, C Reis… - Brain, behavior, and …, 2018 - ncbi.nlm.nih.gov
Neuroinflammation is an essential mechanism involved in the pathogenesis of
subarachnoid hemorrhage (SAH)-induced brain injury. Recently, Netrin-1 (NTN-1) is well …

Recombinant Netrin-1 binding UNC5B receptor attenuates neuroinflammation and brain injury via PPARγ/NFκB signaling pathway after subarachnoid hemorrhage in …

Z Xie, L Huang, B Enkhjargal… - Brain, Behavior …, 2018 - lomalinda.elsevierpure.com
Neuroinflammation is an essential mechanism involved in the pathogenesis of
subarachnoid hemorrhage (SAH)-induced brain injury. Recently, Netrin-1 (NTN-1) is well …

Recombinant Netrin-1 binding UNC5B receptor attenuates neuroinflammation and brain injury via PPARγ/NFκB signaling pathway after subarachnoid hemorrhage in …

Z Xie, L Huang, B Enkhjargal, C Reis… - Brain, Behavior, and …, 2017 - europepmc.org
Neuroinflammation is an essential mechanism involved in the pathogenesis of
subarachnoid hemorrhage (SAH)-induced brain injury. Recently, Netrin-1 (NTN-1) is well …