Exosomes of oral squamous cell carcinoma cells containing miR-181a-3p induce muscle cell atrophy and apoptosis by transmissible endoplasmic reticulum stress …

L Qiu, W Chen, C Wu, Y Yuan, Y Li - Biochemical and Biophysical …, 2020 - Elsevier
Muscle atrophy is a major character of cancer cachexia, whose mechanism remains
enigmatic. During cancer cachexia, the function of endoplasmic reticulum stress (ERS) …

Exosomes of oral squamous cell carcinoma cells containing miR-181a-3p induce muscle cell atrophy and apoptosis by transmissible endoplasmic reticulum stress …

L Qiu, W Chen, C Wu, Y Yuan… - Biochemical and …, 2020 - pubmed.ncbi.nlm.nih.gov
Muscle atrophy is a major character of cancer cachexia, whose mechanism remains
enigmatic. During cancer cachexia, the function of endoplasmic reticulum stress (ERS) …

Exosomes of oral squamous cell carcinoma cells containing miR-181a-3p induce muscle cell atrophy and apoptosis by transmissible endoplasmic reticulum stress …

L Qiu, W Chen, C Wu, Y Yuan, Y Li - Biochemical and Biophysical …, 2020 - europepmc.org
Muscle atrophy is a major character of cancer cachexia, whose mechanism remains
enigmatic. During cancer cachexia, the function of endoplasmic reticulum stress (ERS) …

[引用][C] Exosomes of oral squamous cell carcinoma cells containing miR-181a-3p induce muscle cell atrophy and apoptosis by transmissible endoplasmic reticulum …

L Qiu, W Chen, C Wu, Y Yuan, Y Li - 2020