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[HTML][HTML] PRMT1 methylates METTL14 to modulate its oncogenic function

J Wang, Z Wang, H Inuzuka, W Wei, J Liu - Neoplasia, 2023 - Elsevier
Abstract N6-methyladenosine (m6A), the most abundant mRNA modification in mammalian
cells, is responsible for mRNA stability and alternative splicing. The METTL3-METTL14 …
被引用次数：7 相关文章

PRMT1 methylates METTL14 to modulate its oncogenic function.

J Wang, Z Wang, H Inuzuka, W Wei… - Neoplasia (New York, NY …, 2023 - europepmc.org
N6-methyladenosine (m6A), the most abundant mRNA modification in mammalian cells, is
responsible for mRNA stability and alternative splicing. The METTL3-METTL14-WTAP …

PRMT1 methylates METTL14 to modulate its oncogenic function

J Wang, Z Wang, H Inuzuka… - Neoplasia (New York …, 2023 - pubmed.ncbi.nlm.nih.gov
N6-methyladenosine (m6A), the most abundant mRNA modification in mammalian cells, is
responsible for mRNA stability and alternative splicing. The METTL3-METTL14-WTAP …
[HTML] nih.gov

[HTML][HTML] PRMT1 methylates METTL14 to modulate its oncogenic function

J Wang, Z Wang, H Inuzuka, W Wei… - Neoplasia (New York, NY …, 2023 - ncbi.nlm.nih.gov
Abstract N6-methyladenosine (m6A), the most abundant mRNA modification in mammalian
cells, is responsible for mRNA stability and alternative splicing. The METTL3-METTL14 …

[引用][C] PRMT1 methylates METTL14 to modulate its oncogenic function

J Wang, Z Wang, H Inuzuka, W Wei, J Liu - Neoplasia, 2023 - europepmc.org
abstract N6-methyladenosine (m6A), the most abundant mRNA modification in mammalian
cells, is responsible for mRNA stability and alternative splicing. The METTL3-METTL14 …