Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino… - The FASEB …, 2008 - Wiley Online Library
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

[引用][C] Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology inmdxmice

M Nakatani, Y Takehara, H Sugino, M Matsumoto… - The FASEB …, 2007 - cir.nii.ac.jp
Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle
mass and ameliorates dystrophic pathology in<i>mdx</i>mice | CiNii Research CiNii 国立 …

Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino… - FASEB …, 2008 - pure.fujita-hu.ac.jp
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino… - … publication of the …, 2008 - pubmed.ncbi.nlm.nih.gov
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

[PDF][PDF] Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino, M Matsumoto… - researchgate.net
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino… - FASEB …, 2008 - kyushu-u.pure.elsevier.com
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice.

M Nakatani, Y Takehara, H Sugino… - … : Official Publication of …, 2007 - europepmc.org
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino… - FASEB …, 2008 - kyushu-u.elsevierpure.com
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

[PDF][PDF] Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino, M Matsumoto… - academia.edu
Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …

Transgenic expression of a myostatin inhibitor derived from follistatin increases skeletal muscle mass and ameliorates dystrophic pathology in mdx mice

M Nakatani, Y Takehara, H Sugino… - FASEB …, 2008 - pure.fujita-hu.ac.jp
抄録 Myostatin is a potent negative regulator of skeletal muscle growth. Therefore, myostatin
inhibition offers a novel therapeutic strategy for muscular dystrophy by restoring skeletal …