Activation of Nrf2/AREs-mediated antioxidant signalling, and suppression of profibrotic TGF-β1/Smad3 pathway: a promising therapeutic strategy for hepatic fibrosis …
Y Gong, Y Yang - Life sciences, 2020 - Elsevier
Hepatic fibrosis (HF) is a wound-healing response that occurs during chronic liver injury and
features by an excessive accumulation of extracellular matrix (ECM) components. Activation …
features by an excessive accumulation of extracellular matrix (ECM) components. Activation …
Activation of Nrf2/AREs-mediated antioxidant signalling, and suppression of profibrotic TGF-β1/Smad3 pathway: a promising therapeutic strategy for hepatic fibrosis-A …
Y Gong, Y Yang - Life Sciences, 2020 - europepmc.org
Hepatic fibrosis (HF) is a wound-healing response that occurs during chronic liver injury and
features by an excessive accumulation of extracellular matrix (ECM) components. Activation …
features by an excessive accumulation of extracellular matrix (ECM) components. Activation …
Activation of Nrf2/AREs-mediated antioxidant signalling, and suppression of profibrotic TGF-β1/Smad3 pathway: a promising therapeutic strategy for hepatic fibrosis-A …
Y Gong, Y Yang - Life sciences, 2020 - pubmed.ncbi.nlm.nih.gov
Hepatic fibrosis (HF) is a wound-healing response that occurs during chronic liver injury and
features by an excessive accumulation of extracellular matrix (ECM) components. Activation …
features by an excessive accumulation of extracellular matrix (ECM) components. Activation …