The C5a complement activation peptide increases IL-1β and IL-6 release from amyloid-β primed human monocytes: implications for Alzheimer's disease

S O'Barr, NR Cooper - Journal of neuroimmunology, 2000 - Elsevier
Alzheimer's disease (AD) brains contain large numbers of amyloid-β peptide (Aβ) deposits
associated with activated microglia, astrocytes and dystrophic neurites. Activated …
被引用次数：82 相关文章

The C5a complement activation peptide increases IL-1beta and IL-6 release from amyloid-beta primed human monocytes: implications for Alzheimer's disease

S O'Barr, NR Cooper - Journal of neuroimmunology, 2000 - pubmed.ncbi.nlm.nih.gov
Alzheimer's disease (AD) brains contain large numbers of amyloid-beta peptide (Abeta)
deposits associated with activated microglia, astrocytes and dystrophic neurites. Activated …

The C5a complement activation peptide increases IL-1beta and IL-6 release from amyloid-beta primed human monocytes: implications for Alzheimer's disease.

S O'Barr, NR Cooper - Journal of Neuroimmunology, 2000 - europepmc.org
Alzheimer's disease (AD) brains contain large numbers of amyloid-beta peptide (Abeta)
deposits associated with activated microglia, astrocytes and dystrophic neurites. Activated …

The C5a complement activation peptide increases IL-1β and IL-6 release from amyloid-β primed human monocytes: implications for Alzheimer's disease

S O'Barr, NR Cooper - Journal of Neuroimmunology, 2000 - jni-journal.com
Alzheimer's disease (AD) brains contain large numbers of amyloid-β peptide (Aβ) deposits
associated with activated microglia, astrocytes and dystrophic neurites. Activated …

The C5a complement activation peptide increases IL-1β and IL-6 release from amyloid-β primed human monocytes: implications for Alzheimer's disease

S O'Barr, NR Cooper - Journal of Neuroimmunology, 2000 - infona.pl
Alzheimer's disease (AD) brains contain large numbers of amyloid-β peptide (Aβ) deposits
associated with activated microglia, astrocytes and dystrophic neurites. Activated …