Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

C Liang, Q Shao, W Zhang, M Yang… - Human molecular …, 2019 - academic.oup.com
G4C2 repeat expansions in an intron of C9ORF72 cause the most common familial
amyotrophic lateral sclerosis and frontotemporal dementia (collectively, C9ALS/FTD) …
被引用次数：19 相关文章

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models.

C Liang, Q Shao, W Zhang, M Yang… - Human Molecular …, 2019 - search.ebscohost.com
G4C2 repeat expansions in an intron of C9ORF72 cause the most common familial
amyotrophic lateral sclerosis and frontotemporal dementia (collectively, C9ALS/FTD) …

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models.

C Liang, Q Shao, W Zhang, M Yang… - Human Molecular …, 2019 - europepmc.org
G4C2 repeat expansions in an intron of C9ORF72 cause the most common familial
amyotrophic lateral sclerosis and frontotemporal dementia (collectively, C9ALS/FTD) …

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

C Liang, Q Shao, W Zhang, M Yang… - Human molecular …, 2019 - pubmed.ncbi.nlm.nih.gov
G4C2 repeat expansions in an intron of C9ORF72 cause the most common familial
amyotrophic lateral sclerosis and frontotemporal dementia (collectively, C9ALS/FTD) …

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

C Liang, Q Shao, W Zhang, M Yang… - Human Molecular …, 2019 - academic.oup.com
G4C2 repeat expansions in an intron of C9ORF72 cause the most common familial
amyotrophic lateral sclerosis and frontotemporal dementia (collectively, C9ALS/FTD) …
[HTML] nih.gov

[HTML][HTML] Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

C Liang, Q Shao, W Zhang, M Yang… - Human Molecular …, 2019 - ncbi.nlm.nih.gov
G4C2 repeat expansions in an intron of C9ORF72 cause the most common familial
amyotrophic lateral sclerosis and frontotemporal dementia (collectively, C9ALS/FTD) …

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

C Liang, Q Shao, W Zhang, M Yang… - Human Molecular …, 2020 - ncbi.nlm.nih.gov
Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes
axonal swellings and gain of toxicity in C9ALS/FTD mouse models - PMC Back to Top Skip to …

[引用][C] Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

C Liang, Q Shao, W Zhang, M Yang… - Human molecular …, 2020 - pubmed.ncbi.nlm.nih.gov
Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes
axonal swellings and gain of toxicity in C9ALS/FTD mouse models Smcr8 deficiency disrupts …

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models.

C Liang, Q Shao, W Zhang, M Yang… - Human Molecular …, 2020 - europepmc.org
Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal
swellings and gain of toxicity in C9ALS/FTD mouse models. - Abstract - Europe PMC Sign in …