[HTML][HTML] miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF
Q Wu, L Han, W Yan, X Ji, R Han, J Yang, J Yuan… - Scientific reports, 2016 - nature.com
Silicosis is an incurable occupational disease associated with inflammation, fibroblast
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF
Q Wu, L Han, W Yan, X Ji, R Han, J Yang, J Yuan… - Scientific Reports, 2016 - cir.nii.ac.jp
抄録< jats: title> Abstract</jats: title>< jats: p> Silicosis is an incurable occupational disease
associated with inflammation, fibroblast proliferation and the accumulation of extracellular …
associated with inflammation, fibroblast proliferation and the accumulation of extracellular …
[PDF][PDF] miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF
Q Wu, L Han, W Yan, X Ji, R Han, J Yang, J Yuan, C Ni - cyberleninka.org
Results miR-489 is decreased in mouse lung tissues in a model of silica-induced pulmonary
fibrosis. Our previous microarray data have shown that miR-489 levels in mouse lung …
fibrosis. Our previous microarray data have shown that miR-489 levels in mouse lung …
[HTML][HTML] miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF
Q Wu, L Han, W Yan, X Ji, R Han, J Yang, J Yuan… - Scientific …, 2016 - ncbi.nlm.nih.gov
Silicosis is an incurable occupational disease associated with inflammation, fibroblast
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF.
Q Wu, L Han, W Yan, X Ji, R Han, J Yang, J Yuan… - Scientific …, 2016 - europepmc.org
Silicosis is an incurable occupational disease associated with inflammation, fibroblast
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF
Q Wu, L Han, W Yan, X Ji, R Han, J Yang… - Scientific …, 2016 - ui.adsabs.harvard.edu
Silicosis is an incurable occupational disease associated with inflammation, fibroblast
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF
Q Wu, L Han, W Yan, X Ji, R Han, J Yang… - Scientific …, 2016 - pubmed.ncbi.nlm.nih.gov
Silicosis is an incurable occupational disease associated with inflammation, fibroblast
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF.
Q Wu, L Han, W Yan, X Ji, R Han, J Yang, J Yuan… - Scientific …, 2016 - europepmc.org
Silicosis is an incurable occupational disease associated with inflammation, fibroblast
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …
proliferation and the accumulation of extracellular matrix in lung tissues. The dysregulation …