[HTML] frontiersin.org

[HTML][HTML] NLRX1 deletion increases ischemia-reperfusion damage and activates glucose metabolism in mouse heart

H Zhang, Y Xiao, R Nederlof, D Bakker… - Frontiers in …, 2020 - frontiersin.org
Background NOD-like receptors (NLR) are intracellular sensors of the innate immune
system, with the NLRP3 being a pro-inflammatory member that modulates cardiac ischemia …
被引用次数：18 相关文章

NLRX1 Deletion Increases Ischemia-Reperfusion Damage and Activates Glucose Metabolism in Mouse Heart

H Zhang, Y Xiao, R Nederlof… - Frontiers in …, 2020 - researchinformation.amsterdamumc …
Background: NOD-like receptors (NLR) are intracellular sensors of the innate immune
system, with the NLRP3 being a pro-inflammatory member that modulates cardiac ischemia …

NLRX1 Deletion Increases Ischemia-Reperfusion Damage and Activates Glucose Metabolism in Mouse Heart.

H Zhang, Y Xiao, R Nederlof, D Bakker… - Frontiers in …, 2020 - europepmc.org
Background NOD-like receptors (NLR) are intracellular sensors of the innate immune
system, with the NLRP3 being a pro-inflammatory member that modulates cardiac ischemia …

NLRX1 Deletion Increases Ischemia-Reperfusion Damage and Activates Glucose Metabolism in Mouse Heart

H Zhang, Y Xiao, R Nederlof, D Bakker… - Frontiers in …, 2020 - pubmed.ncbi.nlm.nih.gov
Background NOD-like receptors (NLR) are intracellular sensors of the innate immune
system, with the NLRP3 being a pro-inflammatory member that modulates cardiac ischemia …
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[HTML][HTML] NLRX1 Deletion Increases Ischemia-Reperfusion Damage and Activates Glucose Metabolism in Mouse Heart

H Zhang, Y Xiao, R Nederlof, D Bakker… - Frontiers in …, 2020 - ncbi.nlm.nih.gov
Background NOD-like receptors (NLR) are intracellular sensors of the innate immune
system, with the NLRP3 being a pro-inflammatory member that modulates cardiac ischemia …