P2X ion channel receptors and inflammation

G Burnstock - Purinergic signalling, 2016 - Springer
Neuroinflammation limits tissue damage in response to pathogens or injury and promotes
repair. There are two stages of inflammation, initiation and resolution. P2X receptors are …

P2X ion channel receptors and inflammation

G Burnstock - Purinergic Signalling, 2016 - infona.pl
Neuroinflammation limits tissue damage in response to pathogens or injury and promotes
repair. There are two stages of inflammation, initiation and resolution. P2X receptors are …

P2X ion channel receptors and inflammation.

G Burnstock - Purinergic Signalling, 2016 - europepmc.org
Neuroinflammation limits tissue damage in response to pathogens or injury and promotes
repair. There are two stages of inflammation, initiation and resolution. P2X receptors are …

P2X ion channel receptors and inflammation

G Burnstock - Purinergic signalling, 2016 - pubmed.ncbi.nlm.nih.gov
Neuroinflammation limits tissue damage in response to pathogens or injury and promotes
repair. There are two stages of inflammation, initiation and resolution. P2X receptors are …

[HTML][HTML] P2X ion channel receptors and inflammation

G Burnstock - Purinergic Signalling, 2016 - ncbi.nlm.nih.gov
Neuroinflammation limits tissue damage in response to pathogens or injury and promotes
repair. There are two stages of inflammation, initiation and resolution. P2X receptors are …

[引用][C] P2X ion channel receptors and inflammation

G Burnstock - Purinergic Signalling, 2016 - cir.nii.ac.jp
P2X ion channel receptors and inflammation | CiNii Research CiNii 国立情報学研究所 学術
情報ナビゲータ[サイニィ] 詳細へ移動 検索フォームへ移動 論文・データをさがす 大学図書館の本を …

P2X ion channel receptors and inflammation.

G Burnstock - Purinergic Signalling, 2016 - europepmc.org
Neuroinflammation limits tissue damage in response to pathogens or injury and promotes
repair. There are two stages of inflammation, initiation and resolution. P2X receptors are …

[引用][C] P2X ion channel receptors and inflammation

G Burnstock - Purinergic signalling, 2016 - Springer