… compared with the K + -induced elevation of rCBF in the presence of L-NA and SNAP. We
conclude that NO is a modulator of the rCBF elevation to increased extracellular K + concen …

… of nitric oxide (NO) is responsible for maintaining a more dilated state than in its absence
during increases in extracellular K + … have the ability to modulate dilator responses by providing …

… Furthermore, dilation during neuronal activation is enhanced. Simulations show that
potassium release is responsible for the fast onset of vascular response, whereas NO-modulated …

… which NO is produced in response to neuronal activity to modulate CBF and synaptic plasticity.
… Future work will have to assess whether endothelium-derived NO regulates CBF in brain …

… We used mice lacking endothelial or neuronal nitric oxide synthase (NOS) isoforms (eNOS
−/… of CBF, and its modulation by HBO 2 is responsible for the early vasoconstriction responses…

… agent and [K + ] ACSF are involved in the modulation of the … In series VI, the CBF response
to K + after CSD was decreased … extracellular K + rise or serotonin release for the acute CBF …

… In brain IR, excitatory amino acid toxicity may be modulated by endogenous nitric oxide (NO•) …
In our model, an L-NAME–mediated decrease in CBF was not found at a dose of 20 mg/kg. …

… this study, the CBF response was nulled by … CBF response to stimulation showed only minor
transient changes that were not significantly different from the baseline variation in the CBF …

… Background— From animal studies it emerged that nitric oxide is important for the modulation
of CO 2 -mediated cerebral blood flow (CBF chemoregulation) but not for the pressor-…

… that nitric oxide (NO) serves as a permissive factor in the cerebral blood flow response to …
extra-/intracellular K + concentration, and NO has been suggested to modulate the vascular …