… compared with the K + -induced elevation of rCBF in the presence of L-NA and SNAP. We
conclude that NO is a modulator of the rCBF elevation to increased extracellular K + concen …

… of nitric oxide (NO) is responsible for maintaining a more dilated state than in its absence
during increases in extracellular K + … have the ability to modulate dilator responses by providing …

… agent and [K + ] ACSF are involved in the modulation of the … In series VI, the CBF response
to K + after CSD was decreased … extracellular K + rise or serotonin release for the acute CBF …

… We used mice lacking endothelial or neuronal nitric oxide synthase (NOS) isoforms (eNOS
−/… of CBF, and its modulation by HBO 2 is responsible for the early vasoconstriction responses…

… Background— From animal studies it emerged that nitric oxide is important for the modulation
of CO 2 -mediated cerebral blood flow (CBF chemoregulation) but not for the pressor-…

… that nitric oxide (NO) serves as a permissive factor in the cerebral blood flow response to …
extra-/intracellular K + concentration, and NO has been suggested to modulate the vascular …

… In brain IR, excitatory amino acid toxicity may be modulated by endogenous nitric oxide (NO•) …
In our model, an L-NAME–mediated decrease in CBF was not found at a dose of 20 mg/kg. …

… monitoring probably explains why the dialysate NO x levels in the present study correlated
only with the regional CBF measured at the probe site and not with global CBF values. …

… prevents vasodilator responses to CO 2 (15). Inhibition of NOS also decreases CBF under
… role in modulating the excitatory effects of amino acid neurotransmitters in CNS O 2 toxicity. …

… response to nitric oxide. The artery was precontracted submaximalty with hlstamlne. The
concentrations of nitric oxide … Nitric oxide modulates cerebral blood vessel tone and depresses …