In a previous study, heat shock protein 27 was persistently upregulated in ventral motor
neurons following nerve root avulsion or crush. Here, we examined whether the …

抄録 Root avulsion of adult spinal nerves causes the subacute cell loss of motor neurons. To
explore the mechanisms of the elimination of motor neurons, we investigated the expression …

Lumbosacral nerve root avulsion leads to widespread death of neurons in the anterior horn
area of the injured spinal cord, which results in dysfunction in the lower extremities. Heat …

We investigated injury-induced heat shock protein 27 (HSP27) expression and its
association to axonal outgrowth after injury and different nerve repair models in healthy …

Preconditioning sciatic nerve injury enhances axonal regeneration of ascending sensory
neurons after spinal cord injury. A key question is whether direct injury of sensory nerves is …

The expression of the heat shock proteins hsp27 and hsp70 was examined in the spinal
cord and sciatic nerves of developing rats. Using immunohistochemistry, we found that …

Heat shock protein 27 (Hsp27), a molecular chaperone ubiquitously expressed in many cell
types, has been shown to play a role in protecting neurons from cellular stresses. Unlike …

Peripheral nerve injury in neonatal rats results in the death of the majority of the axotomized
sensory neurons by 7 d after injury. In adult animals, however, all sensory neurons survive …

Heat shock proteins (HSPs) are a family of ubiquitously expressed proteins that are up-
regulated in response to a range of stresses and play an important role in cellular defence …

Heat shock proteins (hsps) are induced in a variety of cells following periods of stress, where
they promote cell survival. In this study, we examined the effect of upregulating hsp …