Various inflammatory stimuli are able to modify or even “re-program” the mitochondrial
metabolism that results in generation of reactive oxygen species. In noncommunicable …

Various inflammatory stimuli are able to modify or even “re-program” the mitochondrial
metabolism that results in generation of reactive oxygen species. In noncommunicable …

Neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease,
Huntington's disease, and amyotrophic lateral sclerosis are a heterogeneous group of …

Brains affected by Alzheimer's disease (AD) show a large spectrum of mitochondrial
alterations at both morphological and genetic level. The causal link between β-amyloid (Aβ) …

Alzheimer's disease (AD) is a neurodegenerative disease with reduced cognitive function
due to mitochondrial dysfunction and oxidative stress. Recent studies show that the …

Mitochondria play a pivotal role in bioenergetics and respiratory functions, which are
essential for the numerous biochemical processes underpinning cell viability. Mitochondrial …

Increased amyloid-beta (Aβ) and amyloid precursor protein (APP) in the brains of
Alzheimer's disease (AD) patients are common pathological hallmarks mediating the …

Chronic neuroinflammation serves a key role in the onset and progression of
neurodegenerative disorders. Mitochondria serve as central regulators of …

Mitochondrial dysfunction and neuroinflammation occur in Alzheimer's disease (AD). The
causes of these pathologic lesions remain uncertain, but links between these phenomena …

Alzheimer's disease (AD) is usually manifested in patients with dementia, accompanied by
anxiety and other mental symptoms. Emerging evidence from humans indicates that people …