[HTML][HTML] Homocysteine exposure impairs myocardial resistance to ischaemia reperfusion and oxidative stress
A Almashhadany, D Shackebaei… - Cellular Physiology and …, 2015 - karger.com
Background/Aims: Hyperhomocysteinaemia is recognised as a strong independent risk
factor for developing cardiovascular disease. This study investigated how an acute
homocysteine dose affected cardiac performance during ischaemia reperfusion and
cardiomyocyte contractility and morphology under normal conditions and during oxidative
stress. Methods: Cardiac function was measured in isolated and perfused rat hearts before
and after 40 minutes' global normothermic ischaemia. Where used, 0.1 mM L-homocysteine …
factor for developing cardiovascular disease. This study investigated how an acute
homocysteine dose affected cardiac performance during ischaemia reperfusion and
cardiomyocyte contractility and morphology under normal conditions and during oxidative
stress. Methods: Cardiac function was measured in isolated and perfused rat hearts before
and after 40 minutes' global normothermic ischaemia. Where used, 0.1 mM L-homocysteine …
Homocysteine Exposure Impairs Myocardial Resistance to Ischaemia Reperfusion and Oxidative Stress.
D Shackebaei, T Van der Touw… - Cellular Physiology …, 2015 - search.ebscohost.com
Abstract Background/Aims: Hyperhomocysteinaemia is recognised as a strong independent
risk factor for developing cardiovascular disease. This study investigated how an acute
homocysteine dose affected cardiac performance during ischaemia reperfusion and
cardiomyocyte contractility and morphology under normal conditions and during oxidative
stress. Methods: Cardiac function was measured in isolated and perfused rat hearts before
and after 40 minutes' global normothermic ischaemia. Where used, 0.1 mM L-homocysteine …
risk factor for developing cardiovascular disease. This study investigated how an acute
homocysteine dose affected cardiac performance during ischaemia reperfusion and
cardiomyocyte contractility and morphology under normal conditions and during oxidative
stress. Methods: Cardiac function was measured in isolated and perfused rat hearts before
and after 40 minutes' global normothermic ischaemia. Where used, 0.1 mM L-homocysteine …
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