… brain injury after CA. Based on these findings, we hypothesized that Ac2-26 could activate
eNOS and ameliorate brain injury … the relationship between Ac2-26 and the eNOS pathway …

… whether the effect of Ac2-26 on brain injury after CPB was associated … brain blood–brain
barrier, and reduced brain injury after CPB. To further assess the effect of Ac2-26 on brain injury, …

… Ac2-26 protects against ischemic or hemorrhage brain injury. The present study was to explore
the effect and mechanism of Ac2-26 on brain injury … effect of Ac2-26 on brain injury after …

… agonist Ac2-26 in a mouse model of pneumococcal meningitis. We demonstrate a protective
effect of Ac2-26 in this model which is mediated via the FPR2 receptor. Ac2-26 treatment …

… shown to protect against lung injury 10 , 11 and brain injury induced by reperfusion via the
… in lung injury after CPB, we hypothesized that Ac2-26 could reduce lung injury after CPB …

… that Ac2-26 administered at … –brain barrier leakage and neuronal apoptosis and improving
outcomes at 3 days post-tMCAO/R. The protection was abrogated when mice received Ac2-26 …

… (ARDS), ventilation also leads to ventilator-induced lung injury (VILI). This study aimed to
estimate the effect and mechanism of Annexin A1 peptide (Ac2-26) on VILI in ARDS rats. …

… factor levels contributes to lung injury and apoptosis [27,28]. The results of this study
indicated that Ac2-26 significantly ameliorated lung histological injuries and apoptosis. …

… In summary, we demonstrated that Ac2-26 attenuated IR-induced lung injury. However,
the protective effect of Ac2-26 was mitigated by the presence of Boc2, a nonselective FPR …

… Finally, Ac2-26 treatment reduced CFA-induced … Ac2-26 attenuates pain by inhibiting
astrocyte activation and the production of inflammatory mediators; thus, this work presents Ac2-26 …