During the last decade much effort has been invested in understanding the events that occur
early after SAH. It is now widely accepted that these early events not only participate in the …

Most subarachnoid hemorrhage (SAH) patients exhibit clinical signs of cerebral ischemia at
admission but no angiographic vasospasm. Consequently, the source of early cerebral …

Cerebral vasospasm research has been focused on investigating the mechanisms of
prolonged delayed vasoconstriction of cerebral arteries following subarachnoid hemorrhage …

The hypothesis that the cerebral microcirculation has to be involved in posthemorrhagic
cerebral ischemia was based on experimental studies showing that acutely following SAH …

Increasingly, experimental research in subarachnoid hemorrhage (SAH) has investigated
early brain injury and the microcirculation. A number of pathophysiological changes occur in …

The term vasospasm is commonly used to describe constriction of cerebral blood vessels
after subarachnoid hemorrhage which results in the restriction of blood flow and ischemia in …

Aneurysmal subarachnoid hemorrhage (SAH) remains a serious cerebrovascular disease.
Even if SAH patients survive the initial insults, delayed cerebral ischemia (DCI) may occur at …

Early brain injury (EBI) after subarachnoid hemorrhage (SAH) is characterized by a severe,
cerebral perfusion pressure (CPP)-independent reduction in cerebral blood flow suggesting …

Microcirculatory dysfunction may contribute to delayed cerebral ischemia after subarachnoid
hemorrhage (SAH). Using a prechiasmatic injection model, this study investigated …

Microcirculatory dysfunction may contribute to delayed cerebral ischemia after subarachnoid
hemorrhage (SAH). This study investigated structural changes in microvessels and their …