Abstract Background/Aim Selenium (Se) levels in pregnancy have been linked to
neurobehavioral development of the offspring. DNA methylation is a potential mechanism …

Background In utero arsenic exposure may alter fetal developmental programming by
altering DNA methylation, which may result in a higher risk of disease in later life. We …

This study evaluated the hypothesis that prenatal maternal socioeconomic status (SES)
adversity is associated with DNA methylation in the placenta. SES adversity was defined by …

Selenium (Se) is an essential trace element involved in various biological processes,
including neurodevelopment. Available literature indicates that both Se deficiency and …

Background Prenatal mercury (Hg) exposure is associated with adverse child
neurobehavioral outcomes. Because Hg can interfere with placental functioning and cross …

Maternal educational attainment (MEA) shapes offspring health through multiple potential
pathways. Differential DNA methylation may provide a mechanistic understanding of these …

Background: Arsenic is one of the most commonly encountered environmental toxicants,
and research from model systems has suggested that one mode of its toxic activity may be …

Background Environmental exposures in utero which modify DNA methylation may have a
long-lasting impact on health and disease in offspring. We aimed to identify and replicate …

Abstract Background Cadmium (Cd) and selenium (Se) antagonistically influence redox
balance and apoptotic signaling, with Cd potentially promoting and Se inhibiting oxidative …

Exposure to arsenic early in life has been associated with increased risk of several chronic
diseases and is believed to alter epigenetic programming in utero. In the present study, we …