Epidemiological studies suggest that chronic exposure to air pollution increases
susceptibility to respiratory infections, including tuberculosis in humans. A possible link …

Diesel exhaust particles (DEP) have strong, selective Th2 adjuvant activity when inhaled
with conventional Ags. We used a novel technique for measuring in vivo cytokine production …

Fine particles derived from diesel engines (diesel exhaust particles, DEP) have attracted
attention, since their density in industrial countries seems related to the increased …

There is growing evidence that diesel exhaust particles (DEP) can induce allergic diseases
with increased IgE production and preferential activation of Th2 cells. To clarify the cellular …

Particulate matter, such as diesel exhaust particles (DEPs), modulate adaptive immune
responses in the lung; however, their mechanism of action remains largely unclear …

Inhalation exposure to indoor air pollutants and cigarette smoke increases the risk of
developing tuberculosis (TB). Whether exposure to ambient air pollution particulate matter …

Objective: Epidemiological as well as experimental studies suggest that particulate air
pollutants, including diesel exhaust particles (DEP), may play a role in the recent increase of …

Smoking increases susceptibility to becoming infected with and developing tuberculosis.
Among the components of cigarette smoke, nicotine has been identified as the main …

The biological effects of acute particulate air pollution exposure in host innate immunity
remain obscure and have relied largely on in vitro models. We hypothesized that single …

Inhalation of diesel exhaust particulate (DEP) causes oxidative stress-induced lung
inflammation. This study investigated the protective effects of TF-343, an antioxidant and anti …