Introduction 3. Biological pathways of PM-induced ROS generation 4. PM perturbation of
mitochondrial function 4.1. Mechanism 1: Effects on the mitochondrial electron transfer chain …

Environmental pollution is a major cause of global mortality and burden of disease. All
chemical pollution forms together may be responsible for up to 12 million annual excess …

Research efforts in the past decades have provided insight into the adverse health effects of
air pollution exposure. Exposure to airborne particulate matter is known to impair the …

Exposure to ambient fine particulate matter (PM2. 5) increases the risk of respiratory
disease. Although previous mitochondrial research has provided new information about PM …

Mitochondria can be used as important biomarkers of pollutants on human health, and fine
particulate matter (PM 2.5) has been documented to cause respiratory damage. However …

Particulate matter (PM) had been associated with cardiotoxicity, while the mechanism of
toxicity has yet to be elucidated, with mitochondria dysfunction as a potential candidate. To …

In order to clarify whether the mitochondrial dysfunction is closely related to the cell
homeostasis maintenance after particulate matter (PM 2.5) exposure, oxidative …

Epidemiological studies revealed a sharp increase in respiratory diseases attributed to PM
2.5. However, the underlying mechanisms remain unclear. Evidence suggested …

Exposure to ambient fine particulate matter (< 2.5 μm; PM2. 5) increases the risk of the
physiopathology of vascular diseases. However, the underlying mechanism, particularly the …

Pulmonary fibrosis is known as an incurable lung disorder with irreversible progression of
chronic injury, myofibroblast proliferation, extracellular matrix (ECM) accumulation, and …