In addition to evidence that inhalation of ambient particulate matter (PM) can increase
cardiopulmonary morbidity and mortality, the brain may also constitute a site adversely …

Increased ambient air particulate matter (PM) concentrations are associated with risk for
myocardial infarction, stroke, and arrhythmia, and ultrafine PM (UFPM) might be particularly …

Air pollution is associated with many neurodevelopmental and neurological disorders in
human populations. Rodent models show similar neurotoxic effects of urban air pollution …

Exposure to air particulate matter (PM) present in urban environments have been shown to
induce systemic prooxidant and proinflammatory effects in apolipoprotein E knockout (ApoE …

Background Deleterious consequences of exposure to traffic emissions may derive from
interactions between carbonaceous particulate matter (PM) and gaseous components in a …

Exposure to particulate matter (PM), a component of urban air pollution, may cause adverse
effects in the brain. Although the exact mechanisms involved are unknown, both oxidative …

Epidemiological studies suggest that ultrafine particles contribute to particulate matter-
induced adverse health effects. Interleukin (IL)-8 is an important proinflammatory cytokine in …

Exposure to particulate matter (PM) air pollution produces inflammatory damage to the
cardiopulmonary system. This toxicity appears to be inversely related to the size of the PM …

The etiology of neurodegenerative disorders is at present unknown. However, many of these
disorders are associated with an increase in oxidative and inflammatory events. Although a …

Exposure to particulate matter (PM) is leading to various respiratory health outcomes.
Compared to coarse and fine particles, less is known about the effects of chronic exposure …