Half the members of the KE family suffer from a speech and language disorder caused by a
mutation in the FOXP2 gene. We examined functional brain abnormalities associated with …

That speech and language are innate capacities of the human brain has long been widely
accepted, but only recently has an entry point into the genetic basis of these remarkable …

Mutations in the human FOXP2 gene cause impaired speech development and linguistic
deficits, which have been best characterised in a large pedigree called the KE family. The …

FOXP2, the first gene to have been implicated in a developmental communication disorder,
offers a unique entry point into neuromolecular mechanisms influencing human speech and …

Disruption of FOXP2, a gene encoding a forkhead‐domain transcription factor, causes a
severe developmental disorder of verbal communication, involving profound articulation …

The most well-described example of an inherited speech and language disorder is that
observed in the multigenerational KE family, caused by a heterozygous missense mutation …

An inherited deficit in spoken language has been associated with a mutation in the forkhead
box P2 (FOXP2) gene on chromosome 7. A recent functional magnetic resonance imaging …

The signalling pathways controlling both the evolution and development of language in the
human brain remain unknown. So far, the transcription factor FOXP2 (forkhead box P2) is …

Disruptions of the human FOXP2 gene cause problems with articulation of complex speech
sounds, accompanied by impairment in many aspects of language ability. The FOXP2/Foxp2 …

In 2001, a point mutation in the forkhead box P2 (FOXP2) coding sequence was identified as
the basis of an inherited speech and language disorder suffered by members of the family …