Neisseria gonorrhoeae, the human obligate pathogen responsible for the sexually
transmitted disease gonorrhea, has evolved several mechanisms to evade the host immune …

The human‐adapted organism Neisseria gonorrhoeae is the causative agent of gonorrhoea,
a sexually transmitted infection. It readily colonizes the genital, rectal and nasalpharyngeal …

Optimal innate immune response to infection includes eradication of potential pathogens,
resolution of associated inflammation, and restitution of homeostasis. Phagocytosing human …

The human pathogen Neisseria gonorrhoeae recruits and interacts extensively with
polymorphonuclear leukocytes (PMNs) during infection. N. gonorrhoeae is able to survive …

Neisseria gonorrhoeae (gonococcus) infection results in recruitment of polymorphonuclear
leukocytes (PMNs) to the urethral lumen. Recent work from our laboratory demonstrated that …

Several microbial pathogens can modulate the host apoptotic response to infection, which
may contribute to immune evasion. Various studies have reported that infection with the …

Current data suggest that Neisseria gonorrhoeae is able to suppress the protective immune
response at different levels, such as B and T lymphocytes and antigen-presenting cells. The …

Neisseria gonorrhoeae is the etiological agent of gonorrhoea, which is a sexually
transmitted disease widespread throughout the world. N. gonorrhoeae does not improve …

Symptomatic infection with Neisseria gonorrhoeae (Gc) results in a potent
polymorphonuclear leukocyte (PMN)‐driven inflammatory response, but the mechanisms by …

Background Progesterone may have clinical relevance in females with asymptomatic
gonococcal infections. Aim To investigate the regulatory effect of progesterone on apoptosis …