Microglia-mediated neuroinflammation is important in the pathogenesis of neonatal hypoxic-
ischemic encephalopathy (HIE). This study aimed to investigate the expression of microRNA …

Abstract Background MicroRNA‐410 (miR‐410) has been found to be deregulated in
neonatal hypoxic‐ischemic encephalopathy (HIE). However, the clinical significance and …

Neonatal hypoxic–ischemic encephalopathy (HIE) is a major cause of lifelong disabilities
worldwide, without effective therapies and clear regulatory mechanisms. MicroRNAs …

Aims Neonatal hypoxic-ischemic encephalopathy (HIE) is still an important cause of
neurological dysfunction. At present, there is no reliable biochemical index in clinical …

Neuroinflammation is a major contributor to secondary neuronal injury that accounts for a
significant proportion of final brain cell loss in neonatal hypoxic-ischemic encephalopathy …

Neonatal hypoxic-ischemic encephalopathy (HIE) is major cause of neonatal death or long-
term neurodevelopmental disabilities, which becomes a major practical problem currently in …

Hypoxic-ischaemic encephalopathy (HIE) resulting from perinatal asphyxia or intrapartum-
related events is a common cause of neonatal brain damage death among neonatal brain …

Background The expression level of miR-376c-3p is significantly lower in infants with
neonatal hypoxic-ischemic encephalopathy (HIE) than in healthy infants. However, the …

Perinatal hypoxic-ischemic encephalopathy (HIE) is associated with high neonatal mortality
and permanent neurologic deficit. Recent data suggested microRNAs (miRNAs) may have …

Objective This study is to investigate the role of microRNA (miR)-30b in the pathogenesis of
hypoxic-ischemic encephalopathy (HIE) in neonates. Methods Totally 26 cases of neonatal …