Object. The authors tested the hypotheses that subarachnoid hemorrhage (SAH) leads to
delayed cell death with the participation of apoptotic-like mechanisms and is influenced by …

OBJECTIVE: The mechanisms underlying neural injury in intracerebral hemorrhage (ICH)
remain uncertain. The present two-part study investigated cell death in the region of ICH and …

OBJECTIVE: To study the occurrence of an inflammatory response in the brain after
subarachnoid hemorrhage and its relation to the decrease in acute cerebral blood flow …

Abstract Objects: The major causes of death and disability in subarachnoid hemorrhage
(SAH) may be early brain injury (EBI) and cerebral vasospasm. Although cerebral …

Brain injury begins early after aneurysmal subarachnoid hemorrhage (SAH). Although cell
death via apoptosis and necrosis is known to be present in brain 24h after SAH, it is not …

The overall hypothesis that cell death after intracerebral hemorrhage is mediated in part by
apoptotic mechanisms was tested. Intracerebral hemorrhage was induced in rats using …

OBJECTIVE: To investigate acute pathophysiological changes after subarachnoid
hemorrhage (SAH) in rats and compare endovascular perforation and double blood …

Clinical evidence suggests that factors other than cerebral vasospasm, such as delayed
neuronal and astrocytic cell death, may play a role in the poor prognosis of patients with …

Object. The pathogenesis of delayed ischemic neurological deficits after subarachnoid
hemorrhage has been related to products of hemolysis. Topical brain superfusion of artificial …

OBJECTIVE: To investigate which of three subarachnoid hemorrhage (SAH) models is the
most suitable for studies of pathological and pathophysiological processes after SAH …