Background. Alopecia areata (AA) is an organ‐specific autoimmune disease characterized
by folliculotropic T‐cell infiltrates around anagen‐stage hair follicles. The role of T helper …

Background Alteration in T‐lymphocyte function and cytokines secreted by T‐cell subsets
has been proposed in the immunopathogenesis of alopecia areata (AA). The role of T …

Alopecia areata (AA) has been represented as a restricted T cell-mediated autoimmune
disease. Several studies have shown that cytokines may play an important role in its …

Background: Most evidence supports the role of altered T cell-mediated immunity in the
pathogenesis of alopecia areata (AA). Tough cytokines and chemokines play an important …

Background Alopecia areata (AA) is an autoimmune disease due to aberrant T‐cell
response against hair follicle self‐antigens. Previous studies support the role of Th1 …

Background: Alopecia areata (AA) is a disease characterized by focally, nonscarring hair
loss on the scalp or any hair-bearing surface. The etiology is unknown, although the …

Alopecia areata (AA) has been considered to be supported by an aberrant expression of IFN-
γ as a result of antigen dependent immune response. On the other hand, AA sometimes …

Background Alopecia areata (AA) is an organ‐specific autoimmune disease characterized
by T‐cell infiltrates and cytokine production. T‐helper 17 (Th17) cells are crucially involved …

Background Alopecia areata (AA) has features of both autoimmune and atopic
pathogenesis, but information on the risk of people with AA developing autoimmune and …

Alopecia areata is a type of non-scarring hair loss. The dysregulation of numerous systemic
Th1 (IL-2, IFN-γ, TNF, IL-12, and IL-18), Th2 (IL-4, IL-5, IL-6, IL-9, IL-10, IL-13, IL-17E, IL-31 …