After traumatic brain injury (TBI), large amounts of red blood cells and hemolytic products
are deposited intracranially creating debris in the cerebrospinal fluid (CSF). This debris …

The distribution and clearance of erythrocytes after subarachnoid hemorrhage (SAH) is
poorly understood. We aimed to characterize the distribution of erythrocytes after SAH and …

Understanding the mechanism of edema formation following intracerebral hemorrhage
(ICH) is important for developing treatment protocols for this condition. This study examines …

Background and Purpose—Red blood cell (RBC) lysis contributes to brain edema formation
after intracerebral hemorrhage (ICH), and RBC hemolysate (oxyhemoglobin) has been …

Object. The mechanisms of brain edema formation following spontaneous intracerebral
hemorrhage (ICH) are not well understood. In previous studies, no significant edema …

After a cerebral hemorrhage (intracerebral, subarachnoid, and intraventricular),
extravasated blood contributes to both initial brain injury, via physical disruption and mass …

Subarachnoid hemorrhage (SAH) is characterized by the extravasation of blood into the
subarachnoid space, in which erythrocyte lysis is the primary contributor to cell death and …

Abundant erythrocytes remain and lyse partially in the subarachnoid space after severe
subarachnoid haemorrhage (SAH). But the effect of subarachnoid erythrocyte lysate on …

In this paper we hypothesize a novel mechanism by which brain injury occurs after
intracerebral hemorrhage. We propose the following mechanism:(1) heme that is derived …

Background and purpose Blood that enters the subarachnoid space (SAS) and its
breakdown products are neurotoxic and are the principal inducers of brain injury after …