Aim: The purpose of the present study was to examine the effect of lipopolysaccharide (LPS)
and intermittent hypoxia–ischemia (HI) on brain damage in neonatal rats. Methods: Seven …

Aim: To determine whether inflammation and hypoxic‐ischemic insult (HI) act additively to
cause brain damage in perinatal animals by examining the dose–response effect of …

Objective: To determine whether, and if so how, iNOS expresses and affects brain injury
induced by hypoxia‐ischemia in an immature brain. Material and Methods: Seven‐day‐old …

Aim: We previously reported that lipopolysaccharide (LPS) and hypoxia‐ischemia (HI) act
additively to induce brain damage in the developing rat model. The present study was …

Perinatal brain damage is associated not only with hypoxic-ischemic insults but also with
intrauterine inflammation. A combination of antenatal inflammation and asphyxia increases …

OBJECTIVE: We assessed the long-term effects of perinatal hypoxia-ischemia and
endotoxin on attention and short-and long-term memory in neonatal rats with the use of …

Objective: Inflammatory cytokines, play a central role in the genesis of preterm parturition
and fetal brain injury. Lipopolysaccharide (LPS) may activate cytokine pathways via …

Prenatal exposure to lipopolysaccharide (LPS) has been exploited to simulate brain
disorder in animal model. Prenatal LPS-exposure has shown elevated levels of pro …

Objective To investigate the gene and protein expressions of three isoforms of nitric oxide
synthase (NOS) and gene expression of Caspase-3, and effect of dexamethasone on them …

Aims: In order to investigate the neuropathological effect on the developing rat brain after
intrauterine infection, 2′, 3′-cyclic nucleotide 3′-phosphodiesterase (CNPase) and …