BACKGROUND: Cardiocirculatory arrest (CCA) activates procoagulant pathways. It has also
been reported to inhibit fibrinolysis, resulting in fibrin deposition and further impairment of …

AIM OF THE STUDY: This study investigated the incidence of hyperfibrinolysis upon
emergency department (ED) admission in patients with out of hospital cardiac arrest …

Objectives: Recent data suggest that early increased fibrinolysis may be associated with
unfavorable prognosis in cardiac arrest. The current study aimed to assess whether there is …

Background Animal studies have demonstrated that hemostatic disorders occurring after
cardiac arrest affect outcome. We investigated hemostatic changes during and after …

Thrombolysis is an effective causal therapy for patients suffering from massive pulmonary
embolism or acute myocardial infarction. In more than 70% of patients with cardiac arrest …

Background: The authors investigated whether patients with out-of-hospital cardiac arrest
with an initial low cerebral oxygen level during cardiopulmonary resuscitation are more …

Hypoxia and ischaemia influence blood coagulation and fibrinolysis. This study has been
made to determine whether human cardiopulmonary arrest causes fibrin formation and …

BACKGROUND We developed a hemorrhagic shock animal model to replicate an urban
prehospital setting where resuscitation fluids are limited to assess the effect of saline versus …

BACKGROUND: Despite its central role in early trauma coagulopathy, abnormal fibrinolysis
continues to be poorly understood. Excessive fibrinolysis is a known contributor to mortality …

Hyperfibrinolysis has been identified as a mechanism of trauma coagulopathy associated
with poor outcome. The aim of the study was to create a trauma coagulopathy model (TCM) …