Particulate air pollution (PM10) stimulates alveolar macrophages (AMs) to release immature
granulocytes from the bone marrow (BM) into the circulation. This study was designed to …

Epidemiologic studies have shown an association between the level of ambient particulate
matter< 10 μm (PM10) and cardiopulmonary mortality. We have shown that exposure of …

Studies have shown that exposure to ambient particulate matter is related to an increased
cardiopulmonary morbidity and mortality. The present study was designed to measure the …

Exposure to ambient air pollution particles with a diameter of< 10 μ m (PM10) has been
associated with increased cardiopulmonary morbidity and mortality. We postulate that these …

Recent studies have shown that an increased concentration of ambient particulate matter
(PM10) is related to decreased pulmonary function and respiratory and cardiovascular …

Elevated levels of ambient particulate matter (PM10) have been associated with increased
cardiopulmonary morbidity and mortality. We previously showed that the deposition of …

Exposure to air pollution [particulate matter, particles< 10 μm (PM10)] causes a systemic
inflammatory response that includes stimulation of the bone marrow (BM) and progression of …

The relationship between the level of particulate air pollution (PM 10) and the mortality and
morbidity rates from respiratory and cardiovascular diseases is well established, but the …

Background Studies from our laboratory have shown that human alveolar macrophages
(AM) and bronchial epithelial cells (HBEC) exposed to ambient particles (PM 10) in vitro …

Atmospheric pollution increases cardiopulmonary morbidity and mortality by unexplained
mechanisms. Phagocytosis of fine particles (PM10) by rabbit alveolar macrophages elevates …