Data reviewed here indicate that acute and chronic ethanol ingestion induce a decrease in
the concentration of GSH and an increase in lipoperoxidation in the liver both in …

Alcoholism is a pervasive problem. The aim of the present study was to clarify the effect of
ethanol on the hepatic glutathione antioxidant system in young and elderly rats. Male albino …

Alcohol consumption is associated with a number of toxicological changes in blood and the
oxidant–antioxidant system. The present study was performed to investigate the alcohol …

The present work has investigated the correlation between short-term ethanol administration
and the free radicals production in rat liver. The non-protein-SH and lipid peroxides …

Free radical generation is an important step in the pathogenesis of ethanol-associated liver
injury. Administration of ethanol induces an increase in lipid peroxidation both by enhancing …

Although it is well known that chronic ethanol abuse produces sexual dysfunction and
impaired spermatogenesis, the mechanisms of ethanol‐induced testicular alterations are not …

Alcoholic liver disease is caused mainly by free radicals. Ascorbic acid (AA) and glutathione
(GSH) are the major water-soluble antioxidants in the liver. The impact of AA …

We have studied the effect of exhaustive concentric physical exercise on glutathione redox
status and the possible relationship between blood glutathione oxidation and blood lactate …

Water containing 20% ethanol was given for a period of 3, 6 and 9 weeks to rats, and
changes in hepatic lipid peroxide, glutathione, glutathione peroxidase and glutathione …

The aim of this study was to investigate whether an 8-week treadmill training attenuates
exerciseinduced oxidative stress in rat liver. Male rats were divided into untrained and …