The study of the influence of the age of animals (13 to 53 weeks) on total liver thiobarbituric
acid reactive substances (TBAR) content showed an increase which is maximal in rats of 39 …

Ethanol consumption and cigarette smoking are common in societies worldwide and have
been identified as injurious to human health. This study was undertaken to examine the …

Alcohol-induced oxidative stress is linked to the metabolism of ethanol involving both
microsomal and mitochondrial systems. Ethanol metabolism is directly involved in the …

The effect of chronic and in vitro ethanol exposure on brain oxygen radical formation and
lipid peroxidation was analyzed. Ethanol induces a dose‐dependent increase in lipid …

The oxidation of ethanol by rat liver microsomes is increased after chronic ethanol
consumption. Previous experiments indicated that hydroxyl radicals play a role in the …

Alcohol is a constituent of the diet that is generally taken in on a voluntary basis. The amount
and type of alcohol consumed along with the frequency of alcohol consumption can vary …

Chronic alcohol consumption causes severe hepatic oxidative damage, particularly to old
subjects by decreasing various antioxidant enzymes. In this study, we test the hypothesis …

It is well known that lipid peroxidation increases with age, and alcohol drinking further
exacerbates this damage. The present study determined the effect of regular exercise …

The effects of ethanol administered as a 15% solution in drinking fluid on weight gain,
soluble liver protein and the activity of the three enzymes of oxygen radical metabolism (ie …

The primary metabolic fate of methanol is oxidation to formaldehyde and then to formate by
enzymes of the liver. Cytochrome P-450 and a role for the hydroxyl radical have been …