Feeding calculi producing diet (CPD) to rats for 4 weeks produced calcium oxaltate stones.
Supplementation of sodium citrate to CPD (c-CPD) prevented stone formation. Except …

Feeding calculi producing diet (CPD) to rats for 4 weeks produced calcium oxalate stones
deposition. Supplementation of methionine to CPD (m-CPD) prevented the stone deposition …

Purpose Renal epithelial cell injury by reactive oxygen species is a prerequisite step in the
pathogenesis of urolithiasis, and there is increasing evidence that reactive oxygen species …

The effect of methionine or citrate on antioxidant defense system has been studied in
urolithic rat. Liver weight and its protein concentration did not change in the rats fed with …

Our objective was to evaluate the oxidative stress and renal tubular cell damage in patients
who have renal stones compared to normal subjects. The patients were re-evaluated after 1 …

Background: Urolithiasis is the third most common urological disease with prevalence of
11% and recurrence rate of 50% and has a great impact on the economsically active …

Membrane injury facilitated the fixation of calcium oxalate crystals and subsequent growth
into kidney stones. Oxalate-induced membrane injury was mediated by lipid peroxidation …

Administration of natural antioxidants has been used to protect against nephrolithiasis.
Urolithiasis was induced by ethylene glycol (EG) in Wistar rats. For 4 weeks, group 1 …

The function of lipid peroxidation and the anti-peroxidative enzymes of rat liver and kidney
were investigated under hyperoxaluric and stone forming conditions. The experimental …

Purpose: We investigated the possible mechanism of increased free radicals, the role of
antioxidant enzymes and their correlation with renal tubular damage in the kidney after …