The aim of the present study was to determine whether neuronal nitric oxide synthase
(nNOS)-derived nitric oxide (NO) plays a permissive role in the regulation of cerebral blood …

The nitric oxide synthase (NOS) inhibitors, nitro-L-arginine, its methyl ester, and N-
monomethyl-L-arginine, have been shown to attenuate resting CBF and hypercapnia …

Abstract CHANGES in nitric oxide (NO) concentration and cerebral blood flow (CBF) in the
parietal cortex during hypercapnoea were investigated in anaesthetized rats, using a NO …

We examined whether attenuation of the hypercapnic increase of cerebral blood flow (CBF)
associated with nitric oxide synthase (NOS) inhibition is related to local neuronal or aortic …

The role of nitric oxide (NO) synthesis in the cerebral hyperemic responses to hypercapnia
and hypoxia was investigated in anesthetized rats. Regional CBF (rCBF) measurements …

We examined the effect of nitric oxide synthase (NOS) inhibition and tetrodotoxin (TTX) on
the increase of cerebral blood flow (CBF) in parietal (CoBF) and cerebellar cortex (CeBF) in …

The effect of NG-nitro-L-arginine (L-NNA) on regional cerebral blood flow (rCBF) response
to hypercapnia (5% CO2 inhalation) was studied in urethan-anesthetized wild-type (SV-129) …

Despite the increasing number of publications devoted to the cerebrovascular role of NO, its
precise influence in awake animals is still poorly characterized. The effect of nitric oxide …

Inhibition of nitric oxide (NO) synthesis attenuates the hypercapnic cerebrovasodilation or
the increases in cerebral blood flow (CBF) produced by acetylcholine (ACh), either topically …

We studied the effect of nitro-L-arginine methyl ester (L-NAME), a nitric oxide synthase
(NOS) inhibitor, on the increases in cerebral blood flow (CBF) elicited by stepwise elevations …