The aim of this study was to explore the intracellular mechanisms underlying the
cardiovascular toxicity of air particulate matter (PM) with an aerodynamic diameter of less …

Urban fine particulate matter (PM 2.5) is a deleterious risk factor in the ambient air and is
recognized to exacerbate atherosclerosis. Perivascular adipose tissue (PVAT) secretes a …

Clinical evidence has identified the pulmonary circulation as an important target of air
pollution. It was previously demonstrated that in vitro exposure to fine particulate matter …

The purpose of this study was to examine the direct toxicity of PM2. 5 collected from Beijing
on human umbilical vein endothelial cells (HUVEC). A Cell Counting Kit 8 (CCK8) assay …

Cardiovascular diseases are the leading cause of death worldwide. Meanwhile,
environment air pollution has become a serious health risk factor, contributing to the …

Although the mechanisms linking cardiopulmonary diseases to ambient fine particles (PM2.
5) are still unclear, inflammation and oxidative stress play important roles in PM2. 5-induced …

Abstract Fine particulate matter (PM 2.5) has attracted increasing attention due to its health-
threatening effects. Although numerous studies have investigated the impact of PM 2.5 on …

Short-and long-term exposure to particulate matter (PM) has been associated with
cardiovascular disease (CVD). It is well recognized that oxidative stress is a potential major …

Clinical evidence has shown an elevated myocardial infarction (MI) risk after PM2. 5
(particulate matter< 2.5 μm) exposure. Incident MI may result from rupture of vulnerable …

Fine particulate matter with an aerodynamic diameter less than 2.5 μm (PM2. 5) is a serious
air pollutant associated with health problems. Macrophages play an important role in the …