Epidemiological evidence suggests that fine particulate matter (PM 2.5) in air pollution
promotes the formation of deep venous thrombosis. However, no evidence is available on …

ABSTRACT ЄAmbient fine particulate matter (PM2. 5) could induce cardiovascular diseases
(CVD), but the mechanism remains unknown. To investigate the roles of epidermal growth …

Objective. To investigate the role of CD4+ CD25+ T cells (Tregs) in protecting fine particulate
matter (PM‐) induced inflammatory responses, and its potential mechanisms. Methods …

As an air pollutant, fine particulate matter with a diameter≤ 2.5 μm (PM2. 5) can enter the
body through the respiratory tract and cause adverse cardiovascular effects. Here, the …

Objective—Toll-like receptor 4 (TLR4) plays a major role mediating endotoxin-induced
cellular inflammation and regulates vascular smooth muscle cell (VSMC) proliferation, which …

Abstract Fine particulate matter (PM 2.5) air pollution poses a major risk to human health
worldwide, and absorbed chemicals play a key role in determining the toxicity of PM 2.5 …

Rationale: Epidemiological evidence indicates that exposures to fine particulate matter air
pollution (PM2. 5) contribute to global burden of disease, primarily as a result of increased …

Exposure to ambient fine particulate matter (PM 2.5) has been associated with vascular
diseases in epidemiological studies. We have demonstrated previously that exposure to …

Introduction PM exposure can induce inflammatory and oxidative responses; however,
differences in these adverse effects have been reported depending on the chemical …

Fine particulate matter≤ 2.5 μm (PM2. 5) air pollution is regarded as one of the prominent
risk factors that contributes to morbidity and mortality globally, among which cardiovascular …