Nucleotide oligomerization domain (NOD)-like receptor protein-3 (NLRP3) inflammasome is
a multiprotein complex, which results in the inflammatory response in early brain injury (EBI) …

Neuroinflammation and white matter microstructure damage are important causes of
cognitive impairment after subarachnoid hemorrhage (SAH). Nod-like receptor protein 3 …

Nucleotide oligomerization domain (NOD)-like receptor protein-3 (NLRP3) inflammasome
may intimately contribute to sustaining damage after traumatic brain injury (TBI). This study …

Activation of the NOD-like receptor protein (NLRP3)-inflammasome has been postulated to
mediate inflammatory responses to brain damage during ischemic/reperfusion (I/R) injury …

Background and Purpose—Intracerebral hemorrhage (ICH) is a devastating disease without
effective treatment. As a key component of the innate immune system, the NOD-like receptor …

Background Neuroinflammation is an essential player in many neurological diseases
including traumatic brain injury (TBI). Recent studies have identified that inflammasome …

The nucleotide-binding oligomerization domain (NOD)-like receptor family pyrin domain
containing 3 (NLRP3) inflammasome-mediated inflammatory response has emerged as a …

Aims Brain edema is a common threat to life in ischaemic brain injury. The NLRP3
inflammasome promotes the inflammatory injury after ischaemic stroke. Previous studies …

Background Inflammasome-mediated neuroinflammation plays an important role in the
pathogenesis of early brain injury (EBI) following subarachnoid hemorrhage (SAH). The …

The NLRP3 inflammasome functions as a crucial component of the inflammatory response
in early brain injury (EBI) after subarachnoid hemorrhage (SAH). However, the mechanisms …