Hepatic ischemia-reperfusion injury is reported to be modulated by neutrophils (PMNs). The
adhesion and emigration of PMNs that precede tissue inflammation and necrosis in other …

The role of neutrophil CD11b/CD18 (Mac‐1) adhesion proteins in the pathogenesis of
hepatic reperfusion injury was investigated in an experimental model. Male Fischer rats …

To determine the role of neutrophils in the pathogenesis of hepatic ischemia/reperfusion
injury, livers from male Fischer rats were subjected to 45 min of no‐flow ischemia followed …

Neutrophils are involved in organ damage induced by an excessive acute inflammatory
response after ischemia‐reperfusion, trauma, and sepsis. In addition to causing vascular …

We investigated the hypothesis that CD54 (intercellular adhesion molecule-1) expressed on
hepatocytes will support beta2-integrin (CD18)-dependent adhesion of neutrophils. An in …

Abdominal sepsis was produced by cecal ligation and puncture (CLP) in rats to observe
neutrophil (PMN) migration into the liver and assess the functional alteration in circulating …

Antibodies against cellular adhesion molecules protect against neutrophil-induced hepatic
injury during ischemia-reperfusion and endotoxemia. To test if p2 integrins on neutrophils …

The chronological expression (over 24 h) of two adhesion molecules [intercellular adhesion
molecule-1 (ICAM-1) and CD11b/CD18 (Mac-1)] and the extent of liver damage, including …

Neutrophils play an important role in ischemia-reperfusion (I/R)-induced vascular injury in
the small intestine. Monoclonal antibodies against the leukocyte adhesion glycoprotein …

This study was designed to assess the effect of neutrophil inhibitory factor (NIF), a novel
specific inhibitor of CD11b/CD18 on hepatic leukocyte trafficking by intravital microscopy 5 h …