Hyperthyroidism is characterized by increased vascular relaxation and decreased vascular
contraction and is associated with augmented levels of triiodothyronine (T3) that contribute …

Context Hypothyroidism impairs cardiovascular health and contributes to endothelial
dysfunction with reduced vasodilation. How 3, 5, 3′-triiodothyronine (T3) and its receptors …

Thyroid hormone has a broad effect on cardiovascular system. 3, 3′, 5-triiodo-l-thyronine
(T3), a biologically active form of thyroid hormone, increases cardiac contractility. T3 causes …

Vascular dysfunction associated with hypertension comprises hypercontractility and
impaired vasodilation. We have previously demonstrated that triiodothyronine (T3), the …

Hypothyroidism is associated with elevated levels of serum thyrotropin (TSH), which have
been shown to promote abnormal proliferation of vascular smooth muscle cells and …

Patients with hyperthyroidism exhibit increased risk of development and progression of
cardiac diseases. The activation of the renin–angiotensin system (RAS) has been indirectly …

Abstract Background/Aims: Vascular relaxation caused by Triiodothyronine (T3) involves
direct activation of endothelial cells (EC) and vascular smooth muscle cells (VSMC) …

Thyroid hormone (TH) treatment exerts beneficial effects on the cardiovascular system: it
lowers cholesterol and LDL levels and enhances cardiac contractile function. However, little …

Objective: Hyperthyroidism has pronounced effects on vascular function and endothelium-
dependent relaxation. The aim of the present study was to identify mechanisms underlying …

Objectives: Thyroid hormone (TH) promotes marked effects on the cardiovascular system,
including the development of cardiac hypertrophy. Some studies have demonstrated that the …