Background Novel molecular positron emission tomography (PET) assessments of fibrosis
have not been compared directly to cardiovascular magnetic resonance (CMR), the current …

Background Novel molecular positron emission tomography (PET) assessments of fibrosis
are now available but have not been compared directly to cardiovascular magnetic …

Background Myocardial fibrosis following myocardial infarction (MI) is a key healing
mechanism and involves widespread fibroblast activation. Once activated, fibroblasts …

Abstract Purpose of the Report Combined cardiac 68 Ga-Fibroblast-Activation Protein-alpha
inhibitor (FAPI) positron-emission tomography (PET) and cardiac magnetic resonance …

Aims The aim of this retrospective analysis was to examine the pattern of cardiac 68 Ga-
fibroblast-activation protein-α inhibitor (FAPI) uptake in patients after acute myocardial …

Purpose To assess predictive value of 68Ga-labeled fibroblast activation protein inhibitor-04
([68Ga] Ga-DOTA-FAPI-04) PET/MR for late left ventricular (LV) remodeling in patients with …

Our previous study has demonstrated the feasibility of noninvasive imaging of fibroblast
activation protein (FAP)-expression after myocardial infarction (MI) in MI-territory in a rat …

Myocardial ischemia may induce myocardial fibrosis, a condition that progressively leads to
ventricular remodeling, heightening the risk of heart failure. The timely detection of …

After acute myocardial infarction (AMI), fibroblast activation protein (FAP) upregulation
exceeds the infarct region. We sought further insights into the physiologic relevance by …

Myocardial fibrosis is the heart's common healing response to injury. While initially seeking
to optimize the strength of diseased tissue, fibrosis can become maladaptive, producing stiff …