The suppression of ox-LDL-induced inflammatory cytokine release and apoptosis of HCAECs by long non-coding RNA-MALAT1 via regulating microRNA-155/SOCS1 …

S Li, Y Sun, L Zhong, Z Xiao, M Yang, M Chen… - Nutrition, Metabolism …, 2018 - Elsevier
Background Atherosclerosis is a chronic inflammatory disease. Accumulating evidence
suggests that long non-coding RNAs (lncRNAs) and microRNAs have emerged as critical …

Long noncoding RNA MALAT1 regulates the progression of atherosclerosis by miR-330-5p/NF-κB signal pathway

Z Shi, Z Zheng, X Lin, H Ma - Journal of cardiovascular …, 2021 - journals.lww.com
Long non-coding RNA metastasis-associated lung adenocarcinoma transcript 1 (MALAT1)
was reported to be related to atherosclerosis (AS) progression. However, the underlying …

[HTML][HTML] Long noncoding RNA MALAT1 promotes high glucose-induced inflammation and apoptosis of vascular endothelial cells by regulating miR-361-3p/SOCS3 …

K Huang, X Yu, Y Yu, L Zhang, Y Cen… - International journal of …, 2020 - ncbi.nlm.nih.gov
Vascular complications are the important pathophysiologic manifestations of patients with
diabetes mellitus (DM) and many long non-coding RNAs (LncRNAs) are involved in this …

LncRNA MALAT1 suppression protects endothelium against oxLDL‐induced inflammation via inhibiting expression of MiR‐181b target gene TOX

L Wang, Y Qi, Y Wang, H Tang, Z Li… - Oxidative Medicine …, 2019 - Wiley Online Library
Rare studies have been conducted to investigate the exact interactions between lung
adenocarcinoma transcript 1 (MALAT1), thymocyte selection‐associated high mobility group …

[HTML][HTML] MIAT promotes proliferation and hinders apoptosis by modulating miR-181b/STAT3 axis in ox-LDL-induced atherosclerosis cell models

X Zhong, X Ma, L Zhang, Y Li, Y Li, R He - Biomedicine & Pharmacotherapy, 2018 - Elsevier
Background Plenty of lncRNAs and microRNAs have been identified to be critical mediators
in the progression of atherosclerosis (AS). Myocardial infarction-associated transcript (MIAT) …

LncRNA MALAT1 promotes STAT3-mediated endothelial inflammation by counteracting the function of miR-590

Q Zhou, Q Run, CY Li, XY Xiong, XL Wu - Cytogenetic and genome …, 2020 - karger.com
The excessive production of inflammatory mediators by vascular endothelial cells (ECs)
greatly contributes to the development of atherosclerosis. In this study, we explored the …

Silencing of MEG3 inhibited ox‐LDL‐induced inflammation and apoptosis in macrophages via modulation of the MEG3/miR‐204/CDKN2A regulatory axis

L Yan, Z Liu, H Yin, Z Guo, Q Luo - Cell biology international, 2019 - Wiley Online Library
Atherosclerosis (AS) is one of the most common chronic inflammatory diseases and a major
cause of morbidity and mortality. However, the underlying molecular mechanisms of the …

[HTML][HTML] LncRNA MEG3-derived miR-361-5p regulate vascular smooth muscle cells proliferation and apoptosis by targeting ABCA1

M Wang, C Li, Y Zhang, X Zhou, Y Liu… - American journal of …, 2019 - ncbi.nlm.nih.gov
Background: Atherosclerosis remains a leading cause of cardiology disease worldwide,
which vascular smooth muscle cells (VSMCs) proliferation and apoptosis are involved …

[HTML][HTML] Long non‑coding RNA MALAT1 regulates cholesterol accumulation in ox‑LDL‑induced macrophages via the microRNA‑17‑5p/ABCA1 axis

L Liu, L Tan, J Yao, L Yang - Molecular medicine …, 2020 - spandidos-publications.com
Atherosclerosis (AS), a major cause of cardiovascular disease, has developed into a serious
challenge to the health system. The long non‑coding RNA (lncRNA) metastasis associated …

[HTML][HTML] Downregulation of microRNA‑106a‑5p alleviates ox‑LDL‑mediated endothelial cell injury by targeting STAT3

Y Hu, R Xu, Y He, Z Zhao, X Mao… - Molecular medicine …, 2020 - spandidos-publications.com
The apoptosis of endothelial cells (ECs) induced by oxidized low‑density lipoprotein (ox‑
LDL) is an important contributing factor in the pathogenesis of atherosclerosis. It has been …