The effects of nitric oxide synthase inhibition on brain acidosis, regional cortical blood flow
(rCBF), and NADH redox state were examined using in vivo fluorescence imaging during …

This experiment examined the effects of nitric oxide (NO) synthase inhibition on brain
intracellular pH, regional cortical blood flow, and NADH fluorescence before and during 3 h …

The interaction between nitric oxide (NO·) and focal cerebral ischemia is multifaceted.
Experiments have shown that inhibition of nitric oxide synthase (NOS) either ameliorates or …

We tested the hypothesis that inhibition of nitric oxide synthase (NOS) activity in brain before
ischemia decreases postischemic hyperemia. Pentobarbital-anesthetized piglets underwent …

We investigated the alterations in the stable end products of nitric oxide, ie, nitrate and
nitrite, in the plasma during and after rat focal cerebral ischemia by an automated procedure …

We tested the hypothesis that decreased tonic release of nitric oxide (NO) or a NO-
containing compound, during postischemic delayed hypoperfusion, would result in an …

The role of nitric oxide (NO) in ischemic neuronal injury is unclear. In permanent focal
ischemia models, NO release has been reported to be both neuroprotective, by virtue of …

This study investigated whether the nitric oxide synthase inhibitor NG-nitro-L-arginine-
methyl ester (L-NAME) would alter blood flow and oxygen balance in the ischemic …

The present study aimed to. examine the effects of NCJ)-nitro-L-arginine (LNA) on the early
ischemic neuronal damage (EIND). All the experiments were carried out under general …

Nitric oxide, a potent vasodilator and an inhibitor of platelet aggregation, may be beneficial
in the early stages of focal cerebral ischemia as it may facilitate collateral blood flow to the …