Background: It has been demonstrated that myocardial ischemia/reperfusion (MI/R) causes
renal damage. However, the mechanism underlying this damage in kidneys during …

BACKGROUND: The role of nitric oxide (NO) production because of inducible nitric oxide
synthase (iNOS) in the pathogenesis of renal ischemia/reperfusion (I/R) injury is unclear. In …

Myocardial ischemia-reperfusion (MI/R) may induce renal damage. A rat model of M/IR
injury was established. The left coronary artery was clamped for 30 min, constituting the …

Objectives Acute kidney injury occurs frequently in patients subsequent to coronary artery
revascularization or myocardial ischemia and reperfusion (MIR). Hypotension and excessive …

Background: It is generally believed that reactive oxygen species (ROS) formation and nitric
oxide (NO) generation by the inducible isoform of nitric oxide synthase (iNOS) are the key …

Inhibition of inducible nitric oxide synthase reduces renal ischemia/reperfusion injury.
Background Nitric oxide (NO), produced via inducible nitric oxide synthase (iNOS), is …

BACKGROUND: Nitric oxide (NO), synthesized from l-arginine by the enzyme NO synthase
(NOS) seems to play an ambiguous role during tissue ischemia-reperfusion (I/R) injury. This …

The objectives of this study were to compare the protective influence of exogenous nitric
oxide on renal ischemia reperfusion (I/R) injury with that of the antioxidant vitamins C and E …

Background: ISCHEMIA/reperfusion-induced acute renal failure causes excretory functional
disorders of nephrones. ISCHEMIA/reperfusion injury increases INOS expression in the …

Introduction: Nitric oxide (NO) is a very important peripheral and renal vasodilator derived
from L-arginine in a reaction modulated by nitric oxide synthase (NOS). We here analyze the …