Cerebral blood flow (CBF) is highly regulated by changes in arterial Pco2 and arterial Po2.
Evidence from animal studies indicates that various vasoactive factors, including release of …

The effect of NG-nitro-L-arginine (L-NNA) on regional cerebral blood flow (rCBF) response
to hypercapnia (5% CO2 inhalation) was studied in urethan-anesthetized wild-type (SV-129) …

We examined 1) whether global cerebral blood flow (CBF) would increase across a 6-h bout
of normobaric poikilocapnic hypoxia and be mediated by a larger increase in blood flow in …

We have previously demonstrated that topical cortical application of nitro-L-arginine (L-NA),
a potent inhibitor of nitric oxide (NO) synthesis, attenuates resting cerebral blood flow (CBF) …

We examined the effect of nitric oxide synthase (NOS) inhibition and tetrodotoxin (TTX) on
the increase of cerebral blood flow (CBF) in parietal (CoBF) and cerebellar cortex (CeBF) in …

Epoxyeicosatrienoic acids are cerebral vasodilators produced in astrocytes by cytochrome P-
450 epoxygenase activity. The P-450 inhibitor miconazole attenuates the increase in …

Regional cerebral blood flow (rCBF) is spatially and temporally adjusted to local energy
needs. This coupling involves dilation of vessels both at the site of metabolite exchange and …

Nitric oxide (NO) regulates basal CBF. In a number of animal models NO has been
implicated in the mediation of the regional changes in CBF (rCBF) that accompany neuronal …

Nitric oxide (NO) increases 3′, 5′-cyclic guanosine monophosphate (cGMP) in vascular
smooth muscle and increases cerebral blood flow (CBF). In early stages of cerebral …

We examined whether attenuation of the hypercapnic increase of cerebral blood flow (CBF)
associated with nitric oxide synthase (NOS) inhibition is related to local neuronal or aortic …