We examined whether attenuation of the hypercapnic increase of cerebral blood flow (CBF)
associated with nitric oxide synthase (NOS) inhibition is related to local neuronal or aortic …

The effects of nitric oxide synthase inhibition on brain acidosis, regional cortical blood flow
(rCBF), and NADH redox state were examined using in vivo fluorescence imaging during …

The relationship between systemic arterial pressure (SAP) and neocortical microcirculatory
blood-flow (CBF) in areas of focal cerebral ischemia was studied in 15 spontaneously …

Disturbances in the nitric oxide (NO) vasodilatory pathway have been implicated in acute
vasoconstriction and ischemia after subarachnoid hemorrhage (SAH). The authors …

The importance of nitric oxide (NO) in regulating cerebral blood flow (CBF) remains
unresolved, due in part to methodological approaches, which lack a comprehensive …

Regional cerebral blood flow (rCBF) is directly coupled to neuronal activity; however, the
mediators of this coupling have not been established. The characterization of these …

This experiment examined the effects of nitric oxide (NO) synthase inhibition on brain
intracellular pH, regional cortical blood flow, and NADH fluorescence before and during 3 h …

We tested the hypothesis that decreased tonic release of nitric oxide (NO) or a NO-
containing compound, during postischemic delayed hypoperfusion, would result in an …

Temporary elevations in cortical cerebral blood flow (CBF) accompany cortical spreading
depression (CSD) in anesthetized animals. We tested the hypothesis that nitric oxide (NO) is …

The interaction between nitric oxide (NO·) and focal cerebral ischemia is multifaceted.
Experiments have shown that inhibition of nitric oxide synthase (NOS) either ameliorates or …