The endothelium-dependent relaxation caused by acetylcholine (ACh) in rabbit aorta
segments was reduced by the nitric oxide (NO) synthase inhibitor NG-nitro-l-arginine methyl …

The purpose of this study was to investigate the effects of inhibitors of the Na+, K+-pump and
membrane depolarizing agents on endothelium-dependent relaxation and elevated cyclic …

We compared the effects of inhibiting nitric oxide synthase (NOS), soluble guanylate cyclase
(sGC) and K+ channel activation on dilator responses to acetylcholine (ACh) in rat …

The possible endothelial factors involved in endothelium-dependent relaxations induced by
acetylcholine (ACh) in aorta, mesenteric and femoral arteries of rabbit were analyzed. In …

Endothelium-dependent relaxation is associated with smooth muscle hyperpolarization in
many arteries which may account for relaxation that persists in the presence of nitric oxide …

The contribution of endothelial factors and mechanisms underlying decreased acetylcholine-
induced relaxation and endothelial inhibitory action on phenylephrine-induced contraction …

This study compared the relaxation induced by acetylcholine (ACh) in aortic and mesenteric
arterial rings from Sprague‐Dawley (SD) rats in the presence and absence of inhibitors of …

Studies were designed to determine the extent of the involvement of endothelium-derived
relaxing factor (s) other than nitric oxide (NO) in vascular relaxation in response to …

Both cGMP-dependent and-independent mechanisms have been implicated in the
regulation of vascular tone by NO. We analyzed acetylcholine (ACh)-and NO-induced …

The mechanisms involved in the mechano-inhibitory effects of acetylcholine (ACh) have
been compared with those of sodium nitroprusside (SNP) and cromakalim on the rat isolated …