Subarachnoid hemorrhage (SAH) is a serious medical problem with few effective
pharmacotherapies available, and neuroinflammation has been identified as an important …

Inflammation and apoptosis are two key factors contributing to secondary brain injury after
intracerebral hemorrhage (ICH). In the present study, we explored the neuroprotective role …

Traumatic brain injury (TBI) is associated with cerebral edema, blood brain barrier
breakdown, and neuroinflammation that contribute to the degree of injury severity and …

Aneurysmal subarachnoid hemorrhage (aSAH) is a devastating disease with high morbidity
and mortality. Cerebral vasospasm, which develops in 3–12 days after aneurysm rupture …

Traumatic brain injury (TBI) leads to permanent neurological impairment, and methylene
blue (MB) exerts central nervous system neuroprotective effects. However, only one …

Early brain injury (EBI), following subarachnoid hemorrhage (SAH), includes blood-brain
barrier (BBB) disruption and consequent edema formation. This study aims to evaluate the …

Increasing numbers of patients with spontaneous subarachnoid hemorrhage (SAH) who
recover from surgery and intensive care management still live with cognitive impairment …

Intracerebral haemorrhage (ICH) induces inflammation, which can cause severe secondary
injury. Recent evidence has suggested that magnolol (MG) has a protective effect against …

Neuroinflammation plays a vital role in early brain injury (EBI) following subarachnoid
hemorrhage (SAH). The hypothesis of this study was that activation of melanocortin 1 …

As is known to all, neuroinflammation plays a vital role in early brain injury pathogenesis
following subarachnoid hemorrhage (SAH). It has been shown that rutin have a property of …